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In vivo modeling of diffuse large B cell lymphoma (DLBCL) with the myeloid differentiation primary response gene 88 (MYD88) L265P mutationon

   
@article{TCR10157,
	author = {Vu N. Ngo},
	title = {In vivo modeling of diffuse large B cell lymphoma (DLBCL) with the myeloid differentiation primary response gene 88 (MYD88) L265P mutationon},
	journal = {Translational Cancer Research},
	volume = {5},
	number = {Suppl 4},
	year = {2016},
	keywords = {},
	abstract = {Diffuse large B cell lymphoma (DLBCL) is the most common lymphoid malignancy, accounting for 30–40% of adult lymphomas. It is a difficult-to-treat disease; only 60% of patients with DLBCL can achieve long-term remission with contemporary immune chemotherapy (rituximab, cyclophosphamide, doxorubicin, vincristine, and prednisone; or R-CHOP). Gene expression profiling has identified at least two biologically distinct DLBCL subtypes based on their cell of origin: the germinal center B cell-like (GCB) and the activated B cell-like (ABC) DLBCL (1).},
	issn = {2219-6803},	url = {https://tcr.amegroups.org/article/view/10157}
}